A connected set of genes associated with programmed cell death 1 implicated in controlling the hypersensitive response in maize 2 3
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چکیده
7 Dept. of Plant Pathology, NC State University, Raleigh NC 27695-7616, USA. 8 Botany and Plant Pathology, Purdue University, Lilly Hall, West Lafayette, IN 47907-2054, 9 USA. 10 Department of Computer Science, University of Missouri, Columbia MO 65211, USA. 11 USDA-ARS Plant Science Research Unit, Raleigh NC 27695, USA. 12 Department of Plant and Soil Sciences, University of Delaware, Newark, DE, USA 19716, 13 USA. 14 Dept of Crop Science, NC State University, Raleigh NC 27695-7620, USA. 15 †† Informatics Institute, University of Missouri, Columbia, MO 65211, USA. 16
منابع مشابه
A connected set of genes associated with programmed cell death implicated in controlling the hypersensitive response in maize.
Rp1-D21 is a maize auto-active resistance gene conferring a spontaneous hypersensitive response (HR) of variable severity depending on genetic background. We report an association mapping strategy based on the Mutant Assisted Gene Identification and Characterization approach to identify naturally occurring allelic variants associated with phenotypic variation in HR. Each member of a collection ...
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One way of controlling plant response to various biotic stresses occurs through programmed cell death, this system is known as the hypersensitive reaction. Plant resistance system against stress is stimulated through different chemicals such as salicylic acid. Therfore, experiment was conducted in a completely randomized design with three replications, in order to identifying the expressed pr...
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Much remains unknown of molecular events controlling the plant hypersensitive defense response (HR), a rapid localized cell death that limits pathogen spread and is mediated by resistance (R-) genes. Genetic control of the HR is hard to quantify due to its microscopic and rapid nature. Natural modifiers of the ectopic HR phenotype induced by an aberrant auto-active R-gene (Rp1-D21), were mapped...
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تاریخ انتشار 2012